Many patients on replacement therapy have difficulty to convert
levothyroxine (T4) to triiodothyronine (T3) efficiently, while the body
needs both hormones. This may be due to an overload of the liver with
toxins or a deficiency in zinc, which is required for the conversion of
T4 to T3. There are also other factors that can interfere with the
conversion of T4 to T3, including nutrient depletion and excess iodine,
which are common root causes.
Because of the longstanding controversy regarding whether hypothyroid
patients can be optimally replaced by treatment with T4 alone,
dissatisfaction with the outcome of T4 monotherapy remains high,
numerous studies have addressed potential benefits of combined therapy
of T3 the T4.
T3 added to sufficient T4 dosing to normalize serum T4 and TSH, all rendering T3 levels to at least middle-normal range.
This compound thyroid hormone drug prescribed for replacement therapy
including conditions after resection of the thyroid gland, radioiodine
therapy, and oncological lesions of the organ, or suppressive therapy.
Replacement therapy is prescribed for hypothyroidism of any etiology:
Primary and secondary hypothyroidism, diffuse and mixed euthyroid
goiter, subacute and autoimmune thyroiditis, thyroid cancer (after
surgical treatment), obesity with hypothyroidism.
EFFECTS:
Boost your metabolism.
Strengthening heat production.
Burning fat.
Stimulating effect on the central nervous system.
Reduced need for sleep.
Improving physical performanc.
DOSAGE:
Initially, small doses are prescribed, with an increase to the
therapeutic dose in 3-4 weeks. In patients with cardiac pathology and
over 65 years of age, the doses of thyroid hormones are reduced (the
risk of side effects from the cardiovascular system). In children,
during pregnancy, the need for thyroid hormones increases. To improve
adaptation to treatment with thyroid hormones, it is possible to
prescribe adreno-blockers.
